A recent post of ZUZU's got me thinking about this subject so I thought I'd open a discussion on which is/should be more important.
Systolic is the highest instantaneous pressure that the heartbeat can generate in the largest arteries of the body; diastolic is the lowest that the pressure drops between the systole peaks. If you look at a graph of pressure you see that most of the graph is far closer to the diastolic than the systolic.
So in the past, the consensus was that contol of diastolic was the best standard for good blood pressure management (and judgement about WHETHER to mange at all.)
In recent years, emphasis has been put more and more on SYSTOLIC pressure. The logic is that a STROKE or blown artery, which is probably the most devastating event that BP can generate would occur only at a SYSTOLE, i.e., a hose will burst at it's HIGHEST pressure, not its lowest.
Little by little I think I am spotting a revival of interest in the diastolic as the more important. These swings can make us patients a bit dizzy, eh wot?
Another concept that has reared its head in the last decade is "PULSE PRESSURE" defined as the difference between systolic and diastolic. It is thought to be a measurement of arterial "stiffness" and may be related to the amount of plaque and calcification that had "hardened" the arterial walls. (Any oldies might remember the term "hardeniing of the arteries" which was loosely tossed around to mean everything from heart disease to senility.) Of course a high systolic has the most impact on amplifying this "stiffness."
Here's my take on the matter, which probably differs from some current norms. I think that diastolic control is very important because, it's the ENDLESS, unremitting pressure on the delicate tubules in the kidney that is a very dangerous aspect of blood pressure. It's also the one aspect of blood pressure that is self perpetuating, i.e., hypertension>>kidney damage>>hypertension.
The pulse pressure, while an interesting phenomenon indicating likely vascular damage, is only illustrative. Controlling pulse pressure (basically, systolic control) won't reverse the arterial damage or the actual "stiffness" just the ability to notice it readily...like painting your speedometer black won't make the car go slower, just your ability to determine the speed.
Of course, nobody could deny the huge stroke risk of a systolic over 200, but using a handful of drugs to get a 145 or 150 down to 135 might not be as wise as some practitioners think. There seems to be some evidence that lowering diastolic (inevitable with these drugs) below 80, or even 82 if I remember correctly, shows a detrimental effect on life expectancy (J-curve phenomenon).
How do you feel about the subject of systolic/diastolic? Any interesting study results you've seen? Anything anecdotal?