I don't know the answer. Everyone is different and individual responses to dietary sodium intake vary. Many mechanisms are involved in long-term blood pressure regulation, with the kidneys playing a major role.
Besides other important functions in the body, sodium and chloride help regulate blood pressure and maintain fluid balance. One gram of sodium is equivalent to 2.5 grams of salt (e.g. sodium chloride).
I think that any acute (not chronic) increases in dietary sodium are quickly corrected by our several internal-control mechanisms (especially pressure natriuresis). The increases in blood pressure after a high sodium meal are transient and, if infrequent, do not have a long-term effect on blood pressure.
The amounts of water and sodium ingested and excreted determine the blood volume. Fortunately, the kidneys regulate the amounts of water and sodium lost into the urine, thus keeping the blood volume within a normal range. Large sodium fluctuations in sodium concentrations are thus avoided. When an excessive amount of water or sodium is consumed, the kidneys respond by excreting more water and sodium. So, a balance of blood volume is maintained. Defects in this or other mechanisms result in an increased blood volume. Increased blood volume ultimately leads to an in increased cardiac output and arterial blood pressure.
The time it takes for the kidneys to process the sodium and/or water (pressure natriuresis) depends on a number of factors. It is a complex ralationship.