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Re: magnesium- the cure for arrythmias? It can certainly help a lot!

Re: magnesium- the cure for arrythmias? It can certainly help a lot!

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Posted by Jofe on July 30, 2000 at 17:01:24:

In Reply to: Re: magnesium- the cure for arrythmias? posted by shellym on May 06, 2000 at 20:48:48:


Magnesium smoothes out spasms in the body whether it is muscular or vascular. In fact, I beleie it is common practice for ER docs to start and IV of magnesium on heart attack patients. The problem is, that I would rather increase my magnesium intake before I become critical. I beleive tht Magnesium levels are not very accurate when captured in blood serum. That test only captures a picture of how much magnesium happens to be in the blood at a particular time--it is not necessarily indicative of how much magnesium is getting to your tissues where it is needed. A more helpful test for me to calculate my magnesium levels wa the magnesium challenge. You collect your urine for 24 hours then you take two injections of magnesium and collect the next 24 hours of urine. If your body does not need the magnesium, your will excrete it through your urine. If your body does need it, especially if you are deficient, your body will absorb most of it and only excrete a little. The lab tests the magnesium levels before and after the shots to calculate the amount used by the body. This test told me how much my body needed magnesium. When I took the test, I not only used most of the magnesium and excreted very little, but I felt really good with magnesium on board. I don’t care what my serum levels are or whether I fall within some arbitrary range considered "normal" I feel better with magnesium and my body seems to hold on ti it.
I took magnesium shots for several weeks and also took it orally. After I built up my magnesium levels I could maintain on just the oral. I also do very well with the B complex which is also helpful for my cardiac and migraine symptoms. When I start to become symptomatic, I go back on the shots or I will get a magnesium/B cmplex/ folic acid IV. I personally feel so much better keeping my magnesium and B levels up. My symptoms (arrhythmia’s) and my energy and my migraines are better.(Migraines are also a result of vascular spasms.) I don’t know why I am low in magnesium, (aside from too much calcium in our diets) but looking for that answer is my next step in helping me get better. A word about calcium—calcium and magnesium go to the same receptor site. If you have too much calcium on board, it will prevent the magnesium from getting to the receptor site so even if you have enough magnesium, it will have a hard time getting absorbed. (another reason why blood levels don't count enough in my book) A doctor offered me a calcium blocker for my migraines and cardiac symptoms but rather than block the calcium, I decided to reduce my intake of calcium and upped my magnesium. It has seemed to help. There is a lot of emphasis of calcium today but our bodies must work in balance so I decided to reduce my calcium. I have Mitral Valve Prolaps and am also a medical reporter/journalist. I am writing a book on this subject and while doing some research I happened on this questions and thought I would share some of the information I have collected during my research. I will attached to this response, some information (a medical study) on Magnesium I found that might be interesting to you.

I am not a doctor I am just a patient like you. I am not recommending any therapies to you, only telling you what I've learned and done. As a journalist, I believe that the more information you have, the better so that you can make informed decisions for yourself with the help of your doctor. I have benefited so much from information others have shared in books, on the internet and posted on sites like this one.


Here's a study I found, but it is only one of many such studies I've seen along the way.


It is now becoming clear that a lower than normal dietary intake of Mg can be a strong risk factor for hypertension, cardiac arrhythmias, ischemic heart disease, atherogenesis and sudden cardiac death. Deficits in serum Mg appear often to be associated with arrhythmias, coronary vasospasm and high blood pressure (from the paper's abstract):
Altura BM, Altura BT, Cardiovascular risk factors and magnesium: relationships to atherosclerosis, ischemic heart disease and hypertension, Magnes Trace Elem, 10:182-192, 1991-92.


Magnes Trace Elem (1991-92;10:182-192

Cardiovascular Risk Factors and Magnesium: Relationships to Atherosclerosis, Ischemic Heart Disease and Hypertension

B.M. Altura, B.T. Altura

Department of Physiology, State University of New York Health Science Center at Brooklyn, N.Y., USA

Key Words. Atherogenesis - Coronary vasospasm Bioenergetics, cellular - Dietary Mg intake - Lipid accumulation- Modulation of Ca metabolism in cardiac and vascular muscle
Abstract. Hypertension and atherosclerosis are well-known precursors of ischemic heart disease, stroke and sudden cardiac death. Although there is general agreement that the atheroma is the hallmark of atherosclerosis and is found in coronary obstruction, there is no agreement as to its etiology. It is now becoming clear that a lower than normal dietary intake of Mg can be a strong risk factor for hypertension, cardiac arrhythmias, ischemic heart disease, atherogenesis and sudden cardiac death. Deficits in serum Mg appear often to be associated with arrhythmias, coronary vasospasm and high blood pressure. Experimental animal studies suggest interrelationships between atherogenesis, hypertension (both systemic and pulmonary) and ischemic heart disease. Evidence is accumulating for a role of Mg2+ in the modulation of serum lipids and lipid uptake in macrophages, smooth muscle cells and the arterial wall. Shortfalls in the dietary intake of Mg clearly exist in Western World populations, and men over the age of 65 years, who are at greatest risk for development and death from ischemic heart disease, have the greatest shortfalls in dietary Mg. It is becoming clear that Mg exerts multiple cellular and molecular effects on cardiac and vascular smooth muscle cells which explain its protective actions.
Introduction
Globally, among the leading causes of death, hypertension and atherosclerosis rank at the top of the list. These cardiovascular diseases, obviously, are the forerunners or precursors of ischemic heart disease, stroke and sudden cardiac death. Among mortality and morbidity indices for man, ischemic heart disease ranks at the top of the list. In the industrialized world ischemic heart disease is the leading killer and accounts for approximately 35% of all deaths each year. The incidence of this disorder rises to 80% in people over 70 years of age. The most common cause of death results from insufficient coronary blood flow.
Some deaths can occur rather suddenly, for example, sudden-death ischemic heart disease. Possibly, as many as 40-60% of the latter may occur in the complete absence of any prior atherosclerosis, thrombus formations or cardiac arrhythmias [for review, see 1]. These syndromes are often referred to as nonocclusive sudden-death ischemic heart disease. Other forms of ischemic heart disease can result in death as a consequence of an acute coronary occlusion or ventricular fibrillation, whereas others are still thought to come about from slow, progressive occlusion of coronary vessels over a period of weeks to years.
Although there is general agreement that the atheroma is the hallmark of atherosclerosis and is found in coronary obstruction, there is no agreement at present as to either the characterization of the early intimal changes or their etiology.




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